Background[ edit ] Fig. The rate of maximum O2 uptake O2max depends on cardiac output, O2 extraction and hemoglobin mass. The cardiac output of an athlete is difficult to manipulate during competitions and the distribution of cardiac output is at the maximum rate i. Therefore, the only method to enhance the physical performance left is to increase the O2 content in the artery by enhancing the hemoglobin mass.
Epo blood doping moral objections are reiterated Epo blood doping and most of us agree that those who opt to cheat in order to win a race are nasty culprits who are destroying the fundamentals of sporting endeavour. As rank as doping is, the commentary on the effects of products like EPO often perpetuates sentiment that needs to be reviewed.
Sipping my coffee and observing the social media hurricane raining down on the latest moral transgressor carries more than a faint echo of the inquisition.
Within a few hours of a doping announcement, gigabytes of tweets, statuses and blogs spring forth. A few hours later and all the major media outlets — the ones who put the Tour on page 15 during the race itself — are all over the story.
Of the young men who gave up their lives for their misguided idea of what it takes to win.
The mechanism of these deaths is well known to anyone with more than a passing interest in cycling. Those who forgot to wake up, or to drink enough water, or simply pushed their haematocrit so high that they created a logjam in their arteries were the victims of this era.
Robin Parisotto, developer of the first EPO test, notes that cardiac arrest and or clots caused by blood overly thickened by drug abuse were the culprits and that these cyclists, in their effort to win at all costs, were accomplices.
These are strong words indeed, allegations of murder or suicide that should, if true, not go unheeded. Whilst neither of the above sources reference evidence to support their claims, Dr David Gerrard — an associate professor of sports medicine, chair of the New Zealand Sports Drug Agency and member of the board of the World Anti-doping Agency — does.
In a text entitled clinical sports medicine, the author posits a possible connection between cardiac arrest and EPO.
The post mortem examinations of athletes alleged to have died from EPO are, to my knowledge, never included. Indeed if we include a search of journalistic sources we find even less precision. The number of deaths ranges from half-a-dozen to 40, the young men in question are either from Belgium, the Netherlands, Luxembourg or some combination of northern European nations.
In the absence of such autopsies, we must look for further evidence to substantiate the stories we tell on long group rides and the internet.
Amongst these statistics, cycling did not figure in the top three sports that were being undertaken at the time of death. Various sources suggest that EPO arrived in professional cycling inyears after three of the 17 cyclists in question had retired. If this is true we can also discount the three riders who died in the off-season and the one who was retired, it seems unlikely that either would have taken an acute dose before their death.
It appears that of the remaining riders, seven were amateurs who seem less likely to have had access to the drug. This leaves a young neo-pro and a cyclocross racer. Still, the above analysis is by no means conclusive. The amateurs could have doped, so could the racers in the off-season.
Cited sources return to a piece of empirical research which demonstrated a coincidence between the use of EPO, HGH and sudden death in a single case but no conlusive cause .
Erythropoietin (EPO) is a naturally-occurring hormone, produced by the kidneys, that stimulates the production of red blood cells. This hormone can also be manufactured and injected into the skin or directly into the bloodstream (intravenously). Portugal’s André Cardoso was provisionally suspended by the UCI on the eve of the Tour de France after an out-of-competition doping test came back positive for the blood booster. Since the introduction of rHuEPO on the market, quite a few strategies have been elaborated to fight blood doping. This review describes the strategies, their limitations, and their potential to fight blood doping more efficiently, notably homologous blood transfusion.
This would challenge the theory that sudden death while sleeping could be due to EPO-induced hypertension. Indeed the only significant reduction in life expectancy achieved through EPO abuse in the literature cited by these many condemning sources was seen in a study of mice with a level of HCT above 80 percent where 47 percent was normal.
These results seem less than relevant to cyclists boosting levels to below 50 percent. Sadly for cycle sport, the easy story to tell — and the one people want to hear — is one of poor choices and terrible consequences.
Not the nuanced one of misfortune and the very real possibility that the guilty ones rode away scott-free to 10 more years of success and a six month plea bargain.
The empirical research does support the notion that young, fit athletes can and do die of cardiac abnormalities and related issues. To remove the misfortune from the deaths of these young men and in some cases accuse authorities and widows of a cover up does us a dual disservice.
Firstly we mislead young athletes about the very real dangers that come from exercising without engaging in a thorough physical check up. Secondly we blame innocent young men for their misfortune.
In a world without the certainty of religion and the inquisitions black and white moral judgements, we all need a moral compass. The secular state is not the immoral free for all that was constructed by religious authorities and neither would cycling be any worse without our doping myth.
We need to be very clear that the moral basis for our anti-doping crusade is not the safety of the athletes so much as the drawing of a line on what is pure and what is not.Blood doping is defined by WADA (World Anti-Doping Agency) as the misuse of techniques and/or substances to increases ones red blood cell count.
Most commonly t.
"The fear with blood doping or EPO use is that the blood count gets too high, the blood gets too thick, and it becomes hard for the heart to push the blood around the body or that somehow this. EPO and Blood Doping EPO is a naturally occurring hormone in the body that stimulates the bone marrow to produce more red cells.
Medically, it is given to patients with anemia of chronic disease whose bone marrow is suppressed to help them have more energy and increase daily function.
As rank as doping is, the commentary on the effects of products like EPO often perpetuates sentiment that needs to be reviewed. It’s fair to make allegations but . -using blood doping to alter the hematocrit also affects normal developmental processes for the blood -parameters like the ratio of reticulocyte hematocrit to total hematocrit become altered -models can predict who has used blood doping based on measurements of these types of parameters (indirect detection).
Nov 25, · Soooo, what I'm hoping to get from you is any positives as to why you would consider participating in blood doping or injecting EPO orrrrr, if any of you have done it, I would love to hear your opinions on it, if you'd do it again, why you did it in the first initiativeblog.com you don't want to make it public, by all means, please send me a message.